Resistant starches differentially stimulate Toll‐like receptors and attenuate proinflammatory cytokines in dendritic cells by modulation of intestinal epithelial cells
Miriam Bermúdez-Brito, Christiane Rösch, Henk A. Schols, Marijke M. Faas, Paul de Vos
Molecular Nutrition & Food Research
Abstract
Resistant starch possesses direct signaling capacity on human DCs in a starch-type-dependent manner. IECs regulate these responses. High-maize® 260 skews toward a more regulatory phenotype in coculture systems of DCs, IEC, and T cells.
Extracted Claims
6 claims extracted from this paper into the knowledge graph
High-maize® 260 skews toward regulatory phenotype
“High-maize® 260 skews toward a more regulatory phenotype in coculture systems of DCs, IEC, and T cells.”
Resistant starch attenuates proinflammatory cytokines
“Resistant starches differentially stimulate Toll‐like receptors and attenuate proinflammatory cytokines in dendritic cells by modulation of intestinal epithelial cells.”
Resistant starch stimulates Toll-like receptors
“Resistant starch possesses direct signaling capacity on human DCs in a starch-type-dependent manner.”